FN ISI Export Format VR 1.0 PT J AU Knobel, R Holditch-Davis, D AF Knobel, Robin Holditch-Davis, Diane TI Thermoregulation and heat loss prevention after birth and during neonatal intensive-care unit stabilization of extremely low-birthweight infants SO JOGNN-JOURNAL OF OBSTETRIC GYNECOLOGIC AND NEONATAL NURSING LA English DT Article DE delivery room resuscitation; ELBW infants; hypothermia; neonatal thermoregulation ID PREMATURE-INFANTS; MORTALITY; ASSOCIATION; SURVIVAL; DELIVERY; TRIAL; LIFE AB Extremely low-birthweight infants have inefficient thermoregulation due to immaturity and may exhibit cold body temperatures after birth and during their first 12 hours of life. Hypothermia in these infants can lead to increased morbidity and mortality. Anecdotal notes made during our recent study revealed extremely low-birthweight infants' temperatures decreased with caregiver procedures such as umbilical line insertion, intubations, obtaining chest x-rays, manipulating intravenous lines, repositioning, suctioning, and taking vital signs during the first 12 hours of life. Therefore, nursing interventions should be undertaken to prevent heat loss during these caregiver procedures. Nurses can improve the thermal environment for extremely low-birthweight infants by prewarming the delivery room and placing the infant in a plastic bag up to the neck during delivery room stabilization to prevent heat loss. C1 Univ N Carolina, Sch Nursing, Chapel Hill, NC USA. Duke Univ, Sch Nursing, Durham, NC USA. RP Knobel, R, 408 Long Point Rd, Chocowinity, NC 27817 USA. 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Poole, Stanley D. Hamdan, Ashraf Minton, Patricia A. Sundell, Hakan W. TI Prenatal nicotine exposure transiently alters the lung mechanical response to hypoxia in young lambs SO RESPIRATORY PHYSIOLOGY & NEUROBIOLOGY LA English DT Article DE respiratory physiology; respiratory function tests; nicotine; prenatal exposure; tobacco smoke pollution; hypoxia; sheep ID INFANT-DEATH-SYNDROME; VENTILATORY RESPONSES; CARDIORESPIRATORY RESPONSE; PULMONARY-FUNCTION; CIGARETTE-SMOKING; MATERNAL SMOKING; GENE-EXPRESSION; NEWBORN RAT; SURFACTANT; COTININE AB To test the hypothesis that fetal nicotine exposure alters the lung mechanical response to hypoxia (10% 02) 10 lambs were exposed during the last fetal trimester to a low dose nicotine (LN) and 10 to a moderate dose (MN) (maternal dose 0.5 and 1.5 mg/(kg day) free base, respectively). There were 10 controls (C). At 12 days, minute ventilation increased significantly less in NIN compared with LN but not with C. In contrast to C and LN, MN did not show anticipated increases in dynamic compliance, specific compliance and FRC or decrease in lung resistance but had signs of airway hyperreactivity during hypoxia. Nicotine exposure did not alter the cardiovascular response. These adverse effects decreased with advancing age. In summary, prenatal nicotine exposure alters the lung mechanical response to hypoxia. We speculate that prenatal nicotine-induced alterations of lung mechanics during hypoxia may contribute to an increased vulnerability to hypoxic stress during infancy. (C) 2006 Elsevier B.V. All rights reserved. C1 Vanderbilt Univ, Sch Med, Dept Pediat, Nashville, TN 37232 USA. Gothenburg Univ, Dept Women & Child Hlth, SE-41685 Gothenburg, Sweden. RP Sundell, HW, Vanderbilt Univ, Sch Med, Dept Pediat, B-1220,MCN, Nashville, TN 37232 USA. 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Physiol. Neuro. PD JUN 15 PY 2007 VL 156 IS 3 BP 283 EP 292 PG 10 SC Physiology; Respiratory System GA 161UH UT ISI:000246041200006 ER EF